Abstract
Ushbu obzor maqolada miokard infarktining o‘tkir davrida rivojlanadigan buyrak disfunksiyasini erta tashxislashda buyrak zararlanishining molekulyar biomarkeri hisoblangan kidney injury molecule-1 (KIM-1) ning diagnostik va prognostik ahamiyati tahlil qilingan. O‘tkir miokard infarkti yurak qon otish hajmining keskin pasayishi, gemodinamik buzilishlar, neyrogumoral tizimlar faollashuvi va yallig‘lanish jarayonlari kuchayishi bilan kechib, o‘tkir buyrak shikastlanishining rivojlanishiga olib keladi.
References
1. Bellomo R., Kellum J.A., Ronco C. (2016). Acute kidney injury. The Lancet, 380(9843), 756–766. https://doi.org/10.1016/S0140-6736(11)61454-
2. Bonventre J. V., Yang L. (2020). Kidney injury molecule-1. Current Opinion in Critical Care, 16(6), 556–561. https://doi.org/10.1097/MCC.0b013e32833e6c4a.
3. Bonventre J.V., Zuk A. (2004). Ischemic acute renal failure: an inflammatory disease? Kidney International, 66(2), 480–485. https://doi.org/10.1111/j.1523- 1755.2004.761_2.x.
4. Coca S. G., Yusuf B., Shlipak M. G., Garg A. X., Parikh C. R. (2019). Long-term risk of mortality and other adverse outcomes after acute kidney injury. Kidney International, 75(8), 836–844. https://doi.org/10.1038/ki.2008.705
5. Coca S.G., Yalavarthy R., Concato J., Parikh C.R. (2018). Biomarkers for the diagnosis and risk stratification of acute kidney injury: a systematic review. Kidney International, 73(9), 1008–1016. https://doi.org/10.1038/sj.ki.5002729